The Brain Lesion Responsible for Parkinsonism After Carbon Monoxide Poisoning

The brain lesion responsible for parkinsonism after carbon monoxide poisoning.

BACKGROUND Parkinsonism is a common neurological sequela of carbon monoxide (CO) poisoning, but its pathophysiological mechanism has yet to be clarified. OBJECTIVES To describe a married couple who were both affected by CO poisoning, but only 1 of whom developed CO-induced parkinsonism, and to discuss the possible underlying pathophysiological mechanism of CO-induced parkinsonism by comparing...

Parkinsonism secondary to carbon monoxide poisoning.

Effect of Erythropoietin on Serum Brain-derived Biomarkers after Carbon Monoxide Poisoning in Rats

Objective(s) Erythropoietin has been shown to exert neuroprotective effects in a variety of CNS injury models. Elevation of serum S100β, as a glial damage marker and myelin basic protein (MBP) has been reported to occur in acute carbon monoxide (CO) poisoning. The aim of this study was to evaluate the effect of erythropoietin (EPO) on serum S100β and MBP levels after CO poisoning in rats. Mat...

[Myocardial infarction after carbon monoxide poisoning].

Carbon monoxide (CO) exposure is a common cause of unintentional poisoning. Clinical manifestation usually involve central nervous and cardio-vascular systems as brain and heart are very sensitive to hypoxia. Myocardial infarction pathogenesis during CO intoxication is complex. Generalised tissue hypoxia and increased thrombotic tendency may play an important role. A case of ST-segment elevatio...

Delayed neurological sequelae after carbon monoxide poisoning.